PeptideDB

Tesamorelin

FDA-approved

FDA-approved stabilised GHRH analog

Also known as: TH9507, Egrifta

Tesamorelin is a stabilised GHRH analog and the only growth-hormone secretagogue with FDA approval specifically for reducing excess visceral fat (in HIV-associated lipodystrophy).

Molecular & research data

Sequence
trans-3-hexenoyl-GHRH(1-44) analog
CAS number
218949-48-5
Molecular formula
C221H366N72O67S
Molecular weight
5135.9 g/mol
Half-life
≈26–38 minutes
Primary targets
GHRH receptor
Routes (research)
Subcutaneous
Storage
Lyophilised: −20 °C. Reconstituted: 2–8 °C, use within weeks.

Overview

Tesamorelin is a synthetic analog of growth-hormone-releasing hormone, based on the full natural GHRH(1-44) sequence with a chemical modification at the N-terminus that improves stability against enzymatic breakdown. It is one of the few peptides in this database with a specific FDA approval — marketed as Egrifta for reducing excess visceral fat in people with HIV-associated lipodystrophy.

Mechanism of action

Like other GHRH analogs, Tesamorelin binds the GHRH receptor on the pituitary, stimulating the natural pulsatile release of growth hormone and a downstream rise in IGF-1. The clinically notable consequence is lipolysis — particularly the breakdown of visceral adipose tissue, the metabolically active fat surrounding the organs.

Its stabilised structure gives it a longer functional profile than the short fragment Sermorelin, while still producing a physiological GH pattern rather than the multi-day exposure of CJC-1295 with DAC.

Common research uses

  • Reduction of visceral adipose tissue (its approved indication)
  • Metabolic and body-composition research
  • Investigations into cognitive and other GH/IGF-1-mediated endpoints

Because it is an approved drug, Tesamorelin has a more robust clinical evidence base than most compounds catalogued here.

Compare Tesamorelin

Tesamorelin FAQ

What is Tesamorelin approved for?+

It is FDA-approved (as Egrifta) to reduce excess abdominal (visceral) fat in adults with HIV-associated lipodystrophy.

How does Tesamorelin differ from Sermorelin?+

Both are GHRH analogs, but Tesamorelin is based on the full GHRH(1-44) sequence with an N-terminal modification that improves stability, and it carries a specific approval for visceral fat reduction.

Does Tesamorelin reduce fat directly?+

Indirectly. By raising endogenous growth hormone, it promotes lipolysis, with the most-studied effect being reduction of visceral adipose tissue.

References

  1. [1]Tesamorelin — PubChemPubChem
  2. [2]Tesamorelin — DrugBankDrugBank

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Last reviewed: 2026-06-26. Information is provided for research and educational reference only — see our disclaimer.